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Accordingly, for the protection and restoration of visual acuity, further studies are necessary that focus on the mechanism of disease progression from early non-proliferative to late proliferative DR. Materials and Methods 2. Support Center Support Center. All transactions at YesAsia. Open in a separate window. Arrowheads indicate empty sleeves. Sustained inflammation after pericyte depletion induces irreversible blood-retina barrier breakdown.
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Invest Ophthalmol Vis Sci. A subset of cerebrovascular pericytes originates from mature macrophages in the very early phase of vascular development in CNS. PDGF is a mitogen of glial cells and mesenchymal cells including vascular pericytes [ 2 , 23 ]. National Center for Biotechnology Information , U. Photoreceptor-specific expression of platelet-derived growth factor-B results in traction retinal detachment. Pericytes promote selective vessel regression to regulate vascular patterning. The progress in understanding and treatment of diabetic retinopathy.
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Materials and Methods 2. Appendix A Supplementary data to this article can be found online at https: These may indicate that neural crest-derived pericytes take major part of coverage in retinal blood vessels. Increased expression of placenta growth factor in proliferative diabetic retinopathy. Biochem Biophys Res Commun.
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Sample preparations and all other procedures for western blotting are described elsewhere [ 41 , 52 ]. Cytokine Growth Factor Rev. Skim milk blocked membranes were incubated with primary antibodies described in Supplementary Information, and then were incubated with the appropriate horseradish peroxidase-conjugated secondary antibodies. Plastic roles of pericytes in the blood-retinal barrier. Click here for instruction.
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